果糖进入细胞不需要胰岛素。

所有其他糖类都会触发胰岛素反应。果糖完全绕过这一机制，直接进入肝脏。

肝脏是唯一能大量处理它的器官。

当大量果糖到达肝脏时，肝脏无法控制摄入速度。它通过一种称为从头脂生成的过程迅速将其转化为脂肪。这些脂肪要么被包装成VLDL颗粒，以甘油三酯形式进入血液，要么留在肝脏并开始在那里积累。

这就是低脂饮食导致脂肪肝的原因。不是膳食脂肪。果糖来自那些被宣传为健康的食物，因为它们不含添加脂肪。

食品行业去除了脂肪，并用糖取代它，通常是高果糖玉米糖浆的形式，因为脂肪携带风味，去除它会让食物尝起来像硬纸板。这种替换已经四十年来一直在推动脂肪肝、高甘油三酯和胰岛素抵抗。

膳食脂肪不会导致脂肪肝。果糖会。

两者一直被持续混淆，这种混淆对销售低脂产品的人来说带来了巨大利润。

几十年低脂饮食后，你的肝脏看起来是什么样子？

Fructose does not need insulin to enter the cell.

Every other sugar triggers an insulin response. Fructose bypasses that mechanism entirely and goes straight to the liver.

The liver is the only organ that can process it in significant amounts.

When fructose arrives at the liver in large quantities, the liver has no way to pace the intake. It converts it rapidly into fat through a process called de novo lipogenesis. That fat either gets packaged into VLDL particles and sent into the bloodstream as triglycerides, or it stays in the liver and starts accumulating there.

This is how a low fat diet causes fatty liver. Not dietary fat. Fructose from foods marketed as healthy because they contain no added fat.

The food industry removed fat and replaced it with sugar, often in the form of high fructose corn syrup, because fat carries flavour and removing it makes food taste like cardboard. That swap has been driving fatty liver, high triglycerides and insulin resistance for forty years.

Dietary fat does not cause fatty liver. Fructose does.

The two have been consistently confused and the confusion has been enormously profitable for the people selling low fat products.

What does your liver look like after decades of low fat eating? @LiveAncestral